Volume 30, Issue 1, 2021


DOI: 10.24205/03276716.2020.2030

LxA4 Inhibits LPS Induced Proinflammatory Response and Microglia Polarization through Notch Signaling Pathway


Abstract
Objective: to analyze the inhibitory effect of lipoxin A4 (LXA4) on lipopolysaccharide (LPS) - induced proinflammatory response and microglial polarization through Notch signaling pathway. Methods: mouse microglial BV2 cells were cultured in vitro and divided into control group (serum-free medium), LXA4 group (100nmol /L LXA4), LPS induced group (200ng/ml LPS), LXA4 + LPS group (100nmol / L LXA4 + 200ng / ml LPS), DAPT + LPS group (100nmol/L LXA4+200ng/ml LPS), DAPT + LXA4 + LPS group (10μM DAPT+200ng/ml LPS). RT-PCR, Western blot and ELISA were used to detect the effects of LxA4 on LPS induced inflammatory response, polarization and downstream molecular activity of Notch signaling pathway. Results: the expression of IL-1 β and TNF - α mRNA in LXA4 + LPS group was significantly lower than that in LPS induced group, while IL-10 mRNA expression was significantly higher than that in LPS induced group (P < 0.05). The expression of iNOS mRNA and protein in LXA4 + LPS group was significantly lower than that in LPS induced group, and Argl mRNA and protein expression was higher than that in LPS induced group (P < 0.05). Notch1, Hes1 mRNA and protein expressions in LXA4 + LPS group were significantly lower than those in LPS induced group, and the expression of Hes5mRNA and protein in LXA4 + LPS group was significantly higher than that in LPS induced group (P < 0.05). Compared with DAPT + LPS group and LXA4 + LPS group, iNOSmRNA in DAPT+LXA4+LPS group was significantly increased, and ArglmRNA was significantly decreased (P < 0.05). Compared with DAPT + LPS group and LXA4 + LPS group, the levels of IL-1 β and TNF - α in DAPT+LXA4+LPS group were significantly increased and IL-10 was decreased obviously (P < 0.05). Conclusion: LxA4 can inhibit LPS induced proinflammatory response and cell polarization of microglia, and its mechanism may be related to the regulation of Notch signaling pathway.

Keywords
LXA4; Notch signaling pathway; LPS; Microglia; Proinflammatory response; Polarization

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